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• Research PerspectiveVolume 1, Issue 11pp 949-953

  Paradoxical down-regulation of p16^INK4a mRNA with advancing age in Acute Myeloid Leukemia

  Relevance score: 0.00

  Hendrik J.M. de Jonge, Carolien M. Woolthuis, Eveline S.J.M. de Bont, Gerwin Huls

  Keywords: p16INK4a, aging, Acute Myeloid Leukemia, senescence

  Published in Aging on October 23, 2009

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• Research PaperVolume 6, Issue 3pp 160-175

  MicroRNA-29 induces cellular senescence in aging muscle through multiple signaling pathways

  Relevance score: 0.00

  Zhaoyong Hu, Janet D. Klein, William E. Mitch, Liping Zhang, Ivan Martinez, Xiaonan H. Wang

  Keywords: p50, p16Ink4A, RB, B-myb, sarcopenia, p85, IGF-1

  Published in Aging on March 12, 2014

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• Research PaperVolume 12, Issue 8pp 6700-6732

  Mitochondrial nucleoid remodeling and biogenesis are regulated by the p53-p21^WAF1-PKCζ pathway in p16^INK4a-silenced cells

  Relevance score: 0.00

  Yun Yeong Lee, Yeon Seung Choi, Do Wan Kim, Jae Youn Cheong, Kye Yong Song, Min Sook Ryu, In Kyoung Lim

  Keywords: senescence, p53-p21-PKCζ activation, p16INK4a silence, mitochondria, nucleoid remodeling

  Published in Aging on April 24, 2020

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• Research PaperVolume 10, Issue 6pp 1356-1365

  Aged cells in human skeletal muscle after resistance exercise

  Relevance score: 0.00

  Chi Yang, Ying Jiao, Bing Wei, Zeyi Yang, Jin-Fu Wu, Jorgen Jensen, Wei-Horng Jean, Chih-Yang Huang, Chia-Hua Kuo

  Keywords: anti-aging, inflammation, p16Ink4a, macrophage, whey protein

  Published in Aging on June 27, 2018

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• Research PaperVolume 8, Issue 7pp 1294-1315

  Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells

  Relevance score: 0.00

  Brandon M. Hall, Vitaly Balan, Anatoli S. Gleiberman, Evguenia Strom, Peter Krasnov, Lauren P. Virtuoso, Elena Rydkina, Slavoljub Vujcic, Karina Balan, Ilya Gitlin, Katerina Leonova, Alexander Polinsky, Olga B. Chernova, Andrei V. Gudkov

  Keywords: chronological aging, inflammation, inflammaging, senescence-associated beta-galactosidase, p16INK4a, senescence-associated secretory phenotype, clodronate

  Published in Aging on July 6, 2016

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• Research PaperVolume 9, Issue 8pp 1867-1884

  p16(Ink4a) and senescence-associated β-galactosidase can be induced in macrophages as part of a reversible response to physiological stimuli

  Relevance score: 0.00

  Brandon M. Hall, Vitaly Balan, Anatoli S. Gleiberman, Evguenia Strom, Peter Krasnov, Lauren P. Virtuoso, Elena Rydkina, Slavoljub Vujcic, Karina Balan, Ilya I. Gitlin, Katerina I. Leonova, Camila R. Consiglio, Sandra O. Gollnick, Olga B. Chernova, Andrei V. Gudkov

  Keywords: aging, macrophage, senescent cell, p16(Ink4a), beta-galactosidase

  Published in Aging on August 2, 2017

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