Research Paper|Volume 11, Issue 18|pp 7830—7846 Downregulation of LncRNA-XIST inhibited development of non-small cell lung cancer by activating miR-335/SOD2/ROS signal pathway mediated pyroptotic cell death
Jinglei Liu1,Harbin Medical University, Heilongjiang, China2Department of Medical Oncology, The Third Affiliated Hospital of Harbin Medical University, Heilong Jiang, China, Lei Yao1,Harbin Medical University, Heilongjiang, China3Department of Thoracic Surgery, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China, Mingyan Zhang1,Harbin Medical University, Heilongjiang, China4Department of Laboratory, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China, Ji Jiang1,Harbin Medical University, Heilongjiang, China5Department of nuclear medicine, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China, Maopeng Yang1,Harbin Medical University, Heilongjiang, China2Department of Medical Oncology, The Third Affiliated Hospital of Harbin Medical University, Heilong Jiang, China, Yue Wang6Department of Pharmacology and Toxicology, Wright State University, Dayton, OH 45435, USA
- 1Harbin Medical University, Heilongjiang, China
- 2Department of Medical Oncology, The Third Affiliated Hospital of Harbin Medical University, Heilong Jiang, China
- 3Department of Thoracic Surgery, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China
- 4Department of Laboratory, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China
- 5Department of nuclear medicine, The Third Affiliated Hospital of Harbin Medical University, Heilongjiang, China
- 6Department of Pharmacology and Toxicology, Wright State University, Dayton, OH 45435, USA
Received: June 3, 2019Accepted: September 14, 2019Published: September 25, 2019
Copyright © 2019 Liu et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
