Research Paper Volume 8, Issue 7 pp 1416—1431
Long-term moderate calorie restriction inhibits inflammation without impairing cell-mediated immunity: a randomized controlled trial in non-obese humans
- 1 The Jean Mayer USDA Human Nutrition Research Center on Aging at Tufts University, Boston, MA 02111, USA
- 2 Duke University Medical Center, Durham, NC 27705, USA
- 3 University of Vermont, Burlington, VT 05405, USA
- 4 Department of Medicine, Washington University School of Medicine, St Louis, MO 63110, USA
- 5 Comparative Medicine and Department of Immunobiology, Yale University School of Medicine, New Haven, CT 06510, USA
- 6 Pennington Biomedical Research Center, Baton Rouge, LA 70808, USA
- 7 Baylor College of Medicine and Michael E DeBakey VA Medical Center, Houston, TX 77030, USA
- 8 Department of Clinical and Experimental Sciences, Brescia University School of Medicine, Brescia, Italy
- 9 CEINGE Biotecnologie Avanzate, Napoli, Italy
Received: May 11, 2016 Accepted: June 20, 2016 Published: July 13, 2016
https://doi.org/10.18632/aging.100994How to Cite
Abstract
Calorie restriction (CR) inhibits inflammation and slows aging in many animal species, but in rodents housed in pathogen-free facilities, CR impairs immunity against certain pathogens. However, little is known about the effects of long-term moderate CR on immune function in humans. In this multi-center, randomized clinical trial to determine CR's effect on inflammation and cell-mediated immunity, 218 healthy non-obese adults (20-50 y), were assigned 25% CR (n=143) or an ad-libitum (AL) diet (n=75), and outcomes tested at baseline, 12, and 24 months of CR. CR induced a 10.4% weight loss over the 2-y period. Relative to AL group, CR reduced circulating inflammatory markers, including total WBC and lymphocyte counts, ICAM-1 and leptin. Serum CRP and TNF-α concentrations were about 40% and 50% lower in CR group, respectively. CR had no effect on the delayed-type hypersensitivity skin response or antibody response to vaccines, nor did it cause difference in clinically significant infections. In conclusion, long-term moderate CR without malnutrition induces a significant and persistent inhibition of inflammation without impairing key in vivo indicators of cell-mediated immunity. Given the established role of these pro-inflammatory molecules in the pathogenesis of multiple chronic diseases, these CR-induced adaptations suggest a shift toward a healthy phenotype.