Research Paper Volume 8, Issue 12 pp 3241—3254
Serum protein mediators of dementia and aging proper
- 1 Department of Psychiatry, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
- 2 Department of Medicine, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
- 3 Department of Family and Community Medicine, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
- 4 South Texas Veterans’ Health System Audie L. Murphy Division GRECC, San Antonio, TX 78229, USA
- 5 Department of Psychiatry, The Medical College of Georgia, Augusta, GA 30912, USA
Received: August 31, 2016 Accepted: November 17, 2016 Published: December 3, 2016
https://doi.org/10.18632/aging.101091How to Cite
Abstract
The latent variable “δ” (for “dementia”) appears to be uniquely responsible for the dementing aspects of cognitive impairment. Age, depressive symptoms, gender and the apolipoprotein E (APOE) ε4 allele are independently associated with δ. In this analysis, we explore serum proteins as potential mediators of age’s specific association with δ in a large, ethnically diverse longitudinal cohort, the Texas Alzheimer’s Research and Care Consortium (TARCC). 22 serum proteins were recognized as partial mediators of age’s association with δ. These include Insulin-like Growth Factor-Binding Protein 2 (IGF-BP2), which we had previously associated with age-specific cognitive change, and both Pancreatic Polypeptide (PP) and von Willebrand Factor (vWF), previously associated with δ. Nine other δ-related proteins were not confirmed by this ethnicity adjusted analysis. Our findings suggest that age’s association with the disabling fraction of cognitive performance is partially mediated by serum proteins, somatomedins and hormones. Those proteins may offer targets for the specific treatment of age-related effects on dementia severity and conversion risk.