Research Paper Volume 3, Issue 4 pp 417—429
Accelerated in vivo epidermal telomere loss in Werner syndrome
- 1 Research Team for Geriatric Pathology, Tokyo Metropolitan Institute of Gerontology, Tokyo 173-0015, Japan
- 2 Department Medical Technology, Faculty of Medical Engineering, Toin University of Yokohama, Yokohama 225-8503, Japan
- 3 Division of Pathology, Cancer Institute, The Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan
- 4 Bioinformatics Group, Genome Center, The Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan
- 5 Department of Dermatology, Dokkyo University School of Medicine, Tochigi 321-0293, Japan
- 6 Department of Pathophysiology, Yokohama College of Pharmacy, Yokohama 245-0066, Japan
Received: April 3, 2011 Accepted: April 23, 2011 Published: April 25, 2011
https://doi.org/10.18632/aging.100315How to Cite
Abstract
Many data pertaining to the accelerated telomere loss in cultured cells derived from Werner syndrome (WS), a representative premature aging syndrome, have been accumulated. However, there have been no definitive data on in vivo telomere shortening in WS patients. In the present study, we measured terminal restriction fragment (TRF) lengths of 10 skin samples collected from extremities of 8 WS patients aged between 30 and 61 years that had been surgically amputated because of skin ulceration, and estimated the annual telomere loss. Whereas the values of TRF length in younger WS patients (in their thirties) were within the normal range, those in older WS patients were markedly shorter relative to non-WS controls. Regression analyses indicated that the TRF length in WS was significantly shorter than that in controls (p < 0.001). Furthermore, we found that TRF lengths in muscle adjacent to the examined epidermis were also significantly shorter than those of controls (p = 0.047). These data demonstrate for the first time that in vivo telomere loss is accelerated in systemic organs of WS patients, suggesting that abnormal telomere erosion is one of the major causes of early onset of age-related symptoms and a predisposition to sarcoma and carcinoma in WS.