Research Perspective Volume 2, Issue 10 pp 731—734
Insights into Cdc13 dependent telomere length regulation
- 1 The Wistar Institute, Gene Expression and Regulation program, 3601 Spruce St, Philadelphia, PA 19103, USA
Received: October 13, 2010 Accepted: October 21, 2010 Published: October 23, 2010
https://doi.org/10.18632/aging.100211How to Cite
Abstract
Cdc13 is a single stranded telomere binding protein that specifically localizes to the telomere ends of budding yeasts and is essential for cell viability. It caps the ends of chromosomes thus preventing chromosome end-to-end fusions and exonucleolytic degradation, events that could lead to genomic instability and senescence, the hallmark of aging. Cdc13 is also involved in telomere length regulation by recruiting or preventing access of telomerase to the telomeric overhang. Recruitment of telomerase to the telomeres for G-strand extension is required for continuous cell division, while preventing its access to the telomeres through capping the chromosome ends prevents mitotic events that could lead to cell immortality, the hall mark of carcinogenesis. Cdc13 and its putative homologues human CTC1 and POT1 are therefore key to many biological processes directly associated with life extension and cancer prevention and can be viewed as an ideal target for cancer and age related therapies.