Research Paper Volume 8, Issue 5 pp 977—985
Defects in MAP1S-mediated autophagy turnover of fibronectin cause renal fibrosis
Figure 2. The impact of MAP1S on the levels of fibronectin in HK2 cells. (A‐D) Representative immunoblot images (A,C) and plots (B,D) showing the impact of MAP1S suppression (A,B) or overexpression (C,D) on the levels of fibronectin in the absence (None) or presence of bafilomycin A1 (BAF). Bars in panels (B,D) represent mean ± standard deviation of fibronectin levels between different groups. The significance is estimated by Student's T Test with two‐tailed distribution and unequal variances. *, p < 0.05; ** and p < 0.01.