Research Paper Volume 1, Issue 8 pp 723—732
Modeling of replicative senescence in hematopoietic development
- 1 Interdisciplinary Center of Scientific Computing (IWR), Institute of Applied Mathematics, University of Heidelberg, 69120 Heidelberg
- 2 Heidelberg Academy of Sciences and Humanities, 69117 Heidelberg, Germany
- 3 Helmholtz Institute for Biomedical Engineering, Aachen University Medical School, 52074 Aachen, Germany
- 4 Department of Medicine V, University of Heidelberg, 69120 Heidelberg, Germany
Received: June 15, 2009 Accepted: July 21, 2009 Published: July 23, 2009
https://doi.org/10.18632/aging.100072How to Cite
Abstract
Hematopoietic stem cells (HSC) give rise to an enormous number of blood cells throughout our life. In contrast their number of cell divisions preceding senescence is limited underin vitro culture conditions. Here we consider the question whether HSC can rejuvenate indefinitely or if the number of cell divisions is restricted. We have developed a multi-compartmental model for hematopoietic differentiation based on ordinary differential equations. The model is based on the hypothesis that in each step of maturation, the percentage of self-renewal versus differentiation is regulated by a single external feedback mechanism. We simulate the model under the assumption that hematopoietic differentiation precedes the six steps of maturation and the cells ultimately cease to proliferate after 50 divisions. Our results demonstrate that it is conceivable to maintain hematopoiesis over a life-time if HSC have a slow division rate and a high self-renewal rate. With age, the feedback signal increases and this enhances self-renewal, which results in the increase of the number of stem and progenitor cells. This study demonstrates that replicative senescence is compatible with life-long hematopoiesis and that model predictions are in line with experimental observations. Thus, HSC might not divide indefinitely with potentially important clinical implications.