Research Paper Volume 16, Issue 19 pp 12909—12927
MIAT promotes myofibroblastic activities and transformation in oral submucous fibrosis through sponging the miR-342-3p/SOX6 axis
Figure 8. A diagram illustrates the mechanism of MIAT in the development of OSF. Upregulation of MIAT in nBMFs following chronic exposure to arecoline may interfere the miR-342-3p-mediated suppression of SOX6, resulting in persistent activation of fBMFs and development of OSF.