Research Paper Volume 16, Issue 7 pp 6588—6612
TGF-β downstream of Smad3 and MAPK signaling antagonistically regulate the viability and partial epithelial–mesenchymal transition of liver progenitor cells
Figure 2. Both Smad and MAPK signaling are activated by TGF-β in liver progenitor cells. (A, B) LPCs (LE/6 and WB-F344 cells) were treated with TGF-β (10 ng/ml) for the indicated times, and the lysates were subjected to Western blot analyses with antibodies against the indicated proteins. Representative blot images of three independent experiments are shown, and GAPDH was used as a loading control.