Research Paper Volume 15, Issue 1 pp 230—245
Increased expression of GPX4 promotes the tumorigenesis of thyroid cancer by inhibiting ferroptosis and predicts poor clinical outcomes
Figure 3. Functional enrichment analysis results. Significant Gene Ontology terms of the GPX4-associated DEGs, including BP (A), CC (B), and MF (C). (D) Significant KEGG pathways of the GPX4-associated DEGs. Significant GSEA results of GPX4-associated DEGs, including Gpcr ligand binding (E), G alpha I signaling events (F), PI3KAKT signaling pathway (G), and pathways in cancer (H).