Figure 9 — S-allyl cysteine reduces osteoarthritis pathology in the tert-butyl hydroperoxide-treated chondrocytes and the destabilization of the medial meniscus model mice via the Nrf2 signaling pathway

Research Paper|Volume 12, Issue 19|pp. 19254–19272

S-allyl cysteine reduces osteoarthritis pathology in the tert-butyl hydroperoxide-treated chondrocytes and the destabilization of the medial meniscus model mice via the Nrf2 signaling pathway

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Figure 9.Schematic diagram shows the potential protective effects of SAC on OA. S-allyl cysteine suppresses senescence and apoptosis of chondrocytes and attenuates extracellular matrix metabolic dysfunction in the osteoarthritis model mice via Nrf2.