Research Paper Volume 16, Issue 13 pp 11090—11102
LIMK1 promotes the development of cervical cancer by up-regulating the ROS/Src-FAK/cofilin signaling pathway
Figure 5. LIMK1 promoted the proliferation of cervical cancer cells. (A) Representative images of SiHa and CaSki cell monoclonal experiments. (B) The results of the clonal formation assay showed that LIMK1 overexpression promoted the formation of cell clones and increased the number of colonies. LIMK1 knockdown inhibited the formation of cell clones, resulting in a decrease in the number of colonies. **P<0.01 indicated statistically significant differences.