Research Paper Volume 14, Issue 19 pp 7877—7889
Hyperlipidemia attenuates the mobilization of endothelial progenitor cells induced by acute myocardial ischemia via VEGF/eNOS/NO/MMP-9 pathway
Figure 9. VEGF, eNOS, and MMP-9 expression in the myocardium of each group rats during 7 days after AMI. Western blot analysis of VEGF (A1), eNOS (B1), and MMP-9 (C1) protein expression in the myocardial tissue of the Ctrl, HL, AMI, and HL-AMI groups. GAPDH was used as the internal reference protein. (A2), (B2), and (C2) show the quantitative analysis of VEGF, eNOS, and MMP-9 respectively. *, **, P < 0.05 or P < 0.01, respectively, compared with the AMI group; #, ##, P < 0.05 or P < 0.01, respectively, compared with the HL group. Each data point represents the average of the group of eight rats, and vertical lines indicate the SEM.